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Univ. Paris-Saclay
Disordered actomyosin contracts in unexpected ways
Martin LENZ
LPTMS CNRS/Paris Sud
Mercredi 07/10/2015, 11:15-12:00
SPEC Salle Itzykson, Bât.774, Orme des Merisiers

The motion of living cells is in large part due to the interaction of semi-flexible actin filaments (F-actin) and myosin molecular motors, which induce the relative sliding of F-actin. It is often assumed that this simple sliding is sufficient to account for all actomyosin-based motion. While this is correct in our highly organized striated muscle, we question the application of this dogma to less ordered actomyosin systems, thus reexamining a cornerstone of our understanding of cellular motion.

Contact : Preden Roulleau

 

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